Static GK- Diseases caused by different Microorganisms – Bacteria, Virus, Fungi, Protozoa – Static GK- Diseases caused by different Microorganisms – diseases caused by viruses, diseases caused by Bacteria,diseases caused by Fungi, Bacterial diseases list, diseases caused by Protozoa. The list of diseases and the microorganisms that cause the disease is as follows –
Diseases caused by Bacteria
 Cholera- Vibrio cholera
 Anthrax- Bacillus Anthraces
 Diphtheria -Corynebacterium diphtheria
 Leprosy – Mycobacterium leprae
 Botulism – Clostridium botulinum
 Syphilis – Treponema pallidum
 Tetanus – Clostridium tetani
 Trachoma -Chlamydia trachomatis
 Tuberculosis -Mycobacterium tuberculosis
 Typhoid fever – Salmonella typhi.
 Whooping cough-Bordetella pertussis
 Diseases caused by Virus
 AIDS-Human Immunodeficiency Virus (HIV)
 Influenza – Influenza virus
 Mumps- Mumps Virus
 Polio -Polio Virus
 Chicken Pox -Varicella zoster virus
 Measles-Measles Virus
 Dengue fever -Dengue Virus
 Chikungunya -Chikungunya virus.
 Rabies -Rabies virus
 SARS – SARS coronavirus
 Diseases caused by Fungi
 Athlete’s foot – caused by the mold Epidermophytonfloccosum
 Diseases caused by Protozoa
 Malaria –   Plasmodium vivax
 Amoebic dysentery – Entamoebahistolytica
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The video discusses the GK Topic of Human diseases caused by Microorganisms – Bacteria, Fungi, Virus and Protozoa. All types of questions asked in Competitive exams have been covered. There are tips to memorize the topic easily.

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Health Vocabulary! https://7esl.com/health-healthcare-vocabulary-english/

Learn common names of illnesses and diseases in English, i.e. asthma, a backache, a broken leg, a cold, a cough, an earache, a fever, the flu, a headache…

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Huntington’s disease is a genetic disorder caused by a breakdown of nerve cells in the brain. The disease affects an individual’s ability to move, their mood, and how they think. There’s currently no cure for Huntington’s disease, but there are types of gene therapy approaches that may offer hope for managing or slowing symptoms.

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Learn all about genetic disorders in just a few minutes! Jessica Pamment, professional lecturer at DePaul University, details the diseases that result from mutations in damaged genes or abnormal numbers of chromosomes.

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What if humans could be edited to run faster, jump higher, and think bigger? What if disease could be eradicated before it ever came to be? These are the questions that no longer belong in just comic books, but in the laboratory of the Innovative Genomics Institute’s Scientific Director, Jacob Corn. In this talk, he explores the future and ethics of CRISPR/Cas-9 genome editing.

[AV and event video provided by http://repertoireproductions.com].

Jacob Corn is the Managing Director and Scientific Director of the Innovative Genomics Initiative and faculty at UC Berkeley in the department of Molecular & Cell Biology. Jacob’s research generally bridges reductionist mechanism with cell biology, with the overarching goal of understanding how biophysical properties interact within the cellular environment to shape signaling behavior and how disease arises when these properties go awry. As the director of the IGI, Jacob is committed to pushing the boundaries of next-generation genome editing for transformative insights into fundamental biologies and to laying the groundwork for clinical and commercial applications of the technology.

This talk was given at a TEDx event using the TED conference format but independently organized by a local community. Learn more at http://ted.com/tedx

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To listen to more of David Weatherall’s stories, go to the playlist: https://www.youtube.com/playlist?list=PLVV0r6CmEsFxwQ7NSziz_XdC66gP3lyYj

British scientist David Weatherall (1933-2018) was a world renowned expert on blood diseases, in particular thalassemias, and used his expertise to help in developing countries. He founded the WIMM at Oxford in 1989 and was knighted in 1987. [Listener: Marcus Pembrey]

TRANSCRIPT: Just before I got- left Liverpool, I got a phone call from a technician from a hospital over on the Wirral, saying that, he’d had a blood from a child, a mentally retarded child, and seen some lumps in the red cells and could they be haemoglobin H. A bloomin’ good technician actually. So, I, so and he sent over a sample, and this was clearly mild haemoglobin H disease, and I went over and saw the family and the parents, and it didn’t look kind of quite right. The child- Inherited the pattern in the parents, and I, I didn’t know what to make of this, so like a lot of these things, I put it away in the drawer, and just after moving to Oxford I got a letter, again, out of the blue, from a Colonel Nicholas Bethlenfalvay from some military hospital in the Deep South of the States. He said he’s got a patient that he thought had haemoglobin H disease, and he was mentally retarded, could we help him sort it out. So, I remembered the Liverpool one, and then I saw a paper in some rather obscure haematological journal, which clearly had been rejected by most of the other journals, about a group in Scandinavia where they’d seen a patient with H disease and a mental retardation, and was just thrown into a group of patients with H disease. So I began to wonder, and so we worked up these, we got samples from the Scandinavian family, we worked up these families as best we could, and the mental retardation varied very widely, there were two of them were quite mild, one was quite severe and had dysmorphic features, and, but, and we did the biosynthesis and we obviously at that stage could, I think we could just, I’m not sure whether we were quite ready with Southern Blotting, I think we were, and we couldn’t find alpha thalassemia in some of the parents, that was the problem, oh, at least, not that we could identify at that stage. So I thought, well, should, you know, should we just sit on this, or should we write a paper, and I thought I will write a very cautious paper entitled Mental- ‘Haemoglobin H Disease Mental Retardation – A True Association or A Chance Association?’ And they, we sent it to the ‘New England Journal’, rather cheekily, and of course, Americans don’t like kind of, sexy titles, and they didn’t like the title at all, and they were going to reject it anyway, and I have to say that the paper would not have got published, except David Nathan, who’s on that editorial board, he thought there might be something, he told them they had to publish it, so they did. And of course, once something happens in a journal like that, you start to get sent patients, and they started to come in, and we had a series of very bright post-docs, Andrew Wilkie particularly, and Richard, and two or three others actually at the time, I think Richard came a little later, and they gradually started to take this apart. Andrew was particularly helpful phenotyping because he was, unlike me, a proper geneticist by then, and they did good genetic studies, and collected long series, and then did the genetics, and it became clear there were two types. There was one that was coded on the chromosome 16, usually due to deletions, and then the more interesting one, which was coded by the X chromosome. And then, much to their credit, they isolated the gene, and it’s been a very interesting story, hasn’t it? Indeed. So, but that again, it was just a kind of, having a look at something that doesn’t quite make sense at the beside, kind of approach, but I, I think that’s a good approach actually, sometimes, but anyway.

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Huntington’s disease is a genetic, neurodegenerative disease with a devastating impact on individuals and entire families. Despite knowing the exact cause of Huntington’s disease since 1993, there still remains much unknown about this complex brain disease.

In this animation, Lauren Byrne, a Research Fellow at the UCL Huntington’s Disease Centre shares her personal experience with Huntington’s. She takes us inside the brain to explore the mechanism of this disease leading to the cognitive, behavioural and motor symptoms in Huntington’s.

This is a Roche video developed in collaboration with Ed Wild, also from UCL Huntington’s Disease Centre, George Yohrling from the Huntington’s Disease Society of America, and Roger and Brenda Wylie, a family living with Huntington’s disease.

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Smallpox is the first and only human disease we’ve totally wiped out. However, thanks to breakthroughs made while eradicating smallpox and a number of other creative solutions , we’ve come really close to making a few more diseases a thing of the past.

This episode contains images of disease.

Hosted by: Michael Aranda

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Sources:
https://www.sciencedirect.com/science/article/pii/S1201971213001240
http://www.nejm.org/doi/full/10.1056/NEJMp078089
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5582630/
https://www.cartercenter.org/health/guinea_worm/
https://www.cdc.gov/vaccines/pubs/pinkbook/downloads/dip.pdf
https://www.historyofvaccines.org/timeline?timeline_categories%5B%5D=50
http://www.who.int/immunization/monitoring_surveillance/data/incidence_series.xls
http://www.who.int/immunization/sage/meetings/2017/april/1_Final_report_Clarke_april3.pdf
https://www.cdc.gov/vaccines/vpd/dtap-tdap-td/public/index.html
https://books.google.com/books hl=en&lr=&id=usLEBAAAQBAJ&oi=fnd&pg=PA225&dq=related:YiVuMqJbDjGhfM:scholar.google.com/&ots=_pbhr6TO89&sig=dhWXDxsRniVtWOiNKpisl5QNjFc#v=onepage&q&f=false
https://www.ncbi.nlm.nih.gov/pubmedhealth/PMHT0024459/
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2487030/pdf/bullwho00397-0005.pdf
https://www.ncbi.nlm.nih.gov/pubmed/3297557
http://www.who.int/lymphatic_filariasis/epidemiology/en/
https://www.cdc.gov/dpdx/lymphaticfilariasis/index.html
https://www.sciencedirect.com/science/article/pii/S1201971213001240
http://www.who.int/neglected_diseases/resources/9789240696471/en/

Find out how smallpox became the first (and only) disease to be permanently eradicated through the use of vaccination and isolation to prevent transmission.

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For most of human history, we have sought to treat and cure diseases. But only in recent decades did it become possible to ensure that a particular disease never threatens humanity again. Julie Garon and Walter A. Orenstein detail how the story of smallpox – the first and only disease to be permanently eliminated – shows how disease eradication can happen, and why it is so difficult to achieve.

Lesson by Julie Garon and Walter A. Orenstein, animation by TOGETHER.

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UVA Neurologist Madaline Harrison, MD explains the signs of Huntington’s Disease.

Huntington’s disease is a rare genetic disorder that causes programmed degeneration of brain cells, called neurons, in certain areas of the brain. This talk explores the diagnosis, stages of the disease, and its treatment. The Stanford Huntington’s Disease and Ataxia Clinic has been named a Center of Excellence by the Huntington’s Disease Society of America. This talk reflects the treatment standards offered at Stanford.

Speaker: Veronica Santini, MD Clinical Instructor, Neurology and Neurological Sciences, Stanford University Medical Center

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Inherited Genetic Disorders | Genetics | Biology | FuseSchool

This girl has six fingers on her left hand. She has a condition called polydactyly, which causes extra fingers or toes. Her father and grandmother as well as some of her cousins also have the condition. This should give you a clue about how she came to have it… any ideas? Well, polydactyly is an example of a genetic disorder – it is inherited. You inherit genes from your parents, and you have two copies of each gene – one from your mother and one from your father. Genes come in different forms, called alleles. Alleles may be recessive or dominant.

So going back to Polydactyly… Polydactyly is caused by a dominant allele, shown as a capital P. You only have to have one copy of this allele to have the condition. So in an example the father has a copy of this allele, but the mother does not… Offspring with one dominant allele big P, and one recessive allele little p will also have the condition. Whereas these offspring won’t have the condition. Can you see what their genotype must be…? Little p, little p – so two copies of the recessive allele.

Another example of an inherited disorder is cystic fibrosis. If you want to discover more about cystic fibrosis, and how it affects the body then watch this video [insert link to ‘cystic fibrosis’ video].

Cystic fibrosis is inherited in a different way to polydactyly. Take a look at this genetic diagram and see if you can figure out how it is different.

To have cystic fibrosis, you have to inherit two recessive alleles. The parents in the diagram are both carriers of the recessive allele, little f. They do not have the disorder themselves but they could both pass the recessive allele to their children.

Quite often, people do not know they are carriers of cystic fibrosis until they have a child with the disorder. This can be very upsetting, as it is a serious condition.

Parents are able to make sure they do not have another child with the disorder using embryo screening.

Let’s finish off with a little discussion about what embryo screening is.

Embryo screening can be used to ensure embryo’s do not suffer from inherited genetic disorders.

To do this, the parents would use in-vitro fertilisation, which is also known as IVF.

IVF is where eggs and sperm are mixed in a dish in a lab outside of the body. Each fertilised egg would be left to grow until it reached a few cells big, and then one cell from each embryo is removed and its genes tested to see if it has the genetic disorder. Only unaffected embryos – a maximum of two – would be placed back into the mother’s uterus to grow and hopefully develop into a healthy baby.

There are many issues surrounding embryo screening. One ethical issue is that the unused embryos, potential lives, are destroyed. Also, there are social concerns – IVF is not always successful and this can be upsetting for the parents and their family. Finally there are also economic issues because IVF is an expensive process.

So there we have a few examples of inherited genetic disorders. These are inherited from your parents, resulting from certain alleles your parents have been carriers of.

Sometimes genetic disorders are caused by dominant alleles – like polydactyly so it can be inherited from one parent – and others are caused by recessive alleles – like cystic fibrosis – so both parents need to carry the allele.

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Narration: Dale Bennet
Script: Gemma Young

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